如何在單字之間的空白處嵌入圖像

Question

我希望將圖像「嵌入」到文件的頁面中，但要使用單字之間的空格來實現。

最後，如果你翻閱這些頁面，我希望能看到一個移動的圖像。

我對自己毫無所獲感到驚訝！涉及單色影像的解決方案將是理想的。

只是為我的狂熱粉絲提供一點東西 8)

Answer 1

在進行了一些問答來了解您的意圖後，我發布這個“答案”，既是為了向其他讀者澄清我認為您問題的意圖是什麼，又是為了斷言我認為它不可能成功。在我的 MWE 中，我嘗試在文字河流中放置一個簡單的圓圈。為了幫助向讀者展示該圓圈，我再次呈現結果，其中空白河流被替換為紅色破折號。那為什麼我認為不可行呢？

1) 在我的 MWE 中，河流被誇大了多個空格，但實際上仍然無法辨別，即使用紅色破折號突出顯示也是如此。要在不如此公然誇張的情況下實現這一目標要困難得多。

2）雖然空白河流可能會分散注意力，但我從未聽說過它可以成為觀察的焦點；

3）在我看來，空白的「像素解析度」非常粗糙，即使在整個頁面的過程中，解析度也不足以將空白中的影像表示為除未解析的斑點之外的任何內容。

但無論如何，請有人證明我錯了。

\documentclass{article}
\usepackage{verbatimbox}
\usepackage{xcolor}
\begin{document}
\let\svdash-
\catcode`-=\active
\def\coloron{\def-{\textcolor{red}{\svdash}}}
\begin{verbnobox}[\rmfamily\coloron]
Here is our goal.  It is a test.  What
we are trying     to see is whether or
if an    image can arise   in
the    rivers of  this text.   That
is   to say,  can one see the    circle
that   is formed  in large    rivers of 
my pic?    Maybe if you    squint, one
can just make        it out. Barely.
Then again, maybe not.
~
Below, the relevant rivers are replaced with dashes
~
Here is our goal.  It is a test.  What
we are trying-----to see is whether or
if an----image can arise---in
the----rivers of  this text.---That
is---to say,  can one see the----circle
that---is formed  in large----rivers of 
my pic?----Maybe if you----squint, one
can just make--------it out. Barely.
Then again, maybe not.
\end{verbnobox}
\end{document}

---

如果您確實想嵌入隱藏圖像，並且我建議您提供比您的實驗室夥伴“朋友”所設想的更成熟的消息，那麼有更簡單的方法：

\documentclass{article}
\usepackage{stackengine,xcolor, graphicx}
\begin{document}
Where is the hidden image%
\stackinset{c}{-.2pt}{b}{.3pt}{\scalebox{0.02}{\textcolor{white}{Hi, mom}}}{?}
\end{document}

Answer 2

大部分可以完成 8) 手動調整右邊距是 PITA 並且不是很準確，因此出於教育目的，這裡是最終的輸出，它既證明又反駁了這是可以完成的並且它有效 8)

  \documentclass{article}
  \usepackage{verbatimbox}
  \usepackage{xcolor}
  \begin{document}
  \begin{verbnobox}[\rmfamily]
  target  of  a given  family’s toxin.   Many type II  TAs  target  translational  machinery,
  which  is  highly  conserved  across  all domains of life  (Poole  and  Logan 2005;  Noller
  2004).  DNA synthesis machinery, DNA polymerases  and  primases  are  less  conserved
  (Poole  and  Logan  2005;  Leipe, Aravind,  and  Koonin 1999;  Werner  and  Grohmann
  2011;  Aravind and  Koonin 2001).  Amongst type I  TAs, SymE,  which targets  mRNA
  (Kawano,  Aravind,  and Storz  2007),  would  provide  an  interesting comparison to the
  membrane proteins investigated within this thesis.

Clea   rly, neither toxi   n norantitoxin target  accounts  for  all  differences within  TA
  syste   ms, because families w   ith the same target often contain loci that differ inability
  to c   onfer a PSK effect on a    plasmid  (Szekeres et  al . 2007;  Christensen,  Maenhaut   
  Mich   el, et al. 2004; De Bast,    Mine, and  Van  Melderen  2008;  Wilbaux et al.  2007;
  Fiebig    et al. 2010). Similarly, ty   pe I  and  III  RMs  are not  as mobile and have not
  been sho   wn to induce PSK (Nadere   r et al.  2002;  O’Sullivan  et al.  2000;  Mruk and
  Kobayashi 2014),    but have the same tar   get (DNA) as  type  II  RMs.  Other  factors
  are also important for    PSK, including the le   vels at  which  the  toxin  and  antitoxin
  are expressed, and the rate    at which the toxin and    antitoxin are degraded in the cell
  after plasmid loss (Chapter 5).

TA systems rely on differential d   ecay of  the  toxin  and ant   itoxin  to  induce  PSK
  (Chapter 5). Type II  systems  are ti   ghtly  regulated  by  protein     antitoxins,   which
  interact with the toxin and act as transcr   iptional  repressors,  sensitive    to changes  in
  stoichiometry (Mruk and Kobayashi 2014;    J. Zhang,  Y.  Zhang, and M. In   ouye 2003;
  Kedzierska,  Lian,  and F.  Hayes  2007;    Cataudella, Sneppen, et al. 2013; Cata   udella,
  Trusina,  et  al.  2012; Afif  et al. 2001).    I used  equations  of  logarithmic decay    as a
  starting point for analyzing the condit   ions necessary for type  II  TAs to exhibit P   SK.
  In particular, I analyzed the pop   ulation of toxin and antitoxin in the cell necessary    for
  PSK given their respective half   -lives ( Chapter  5 ). Antitoxins  with similar stabil   ities
  as their associated toxins cannot    be  expressed  in  numbers  significantly  higher    than
  the toxin,  or  there  will  not be su   fficient free  toxin  to create a  PSK effect    within a
  given time period.  Given the  inherent    noise of ge   ne  expression (Rase   r and  O’Shea
  2005; C. V. Rao, D. M. Wolf, and Arkin 2002),    system   s relying    on stable antitoxins
  risk  plasmid  suicide from  excess toxin  in  the cell     prior to  loss. Thus, there  is likely
  to be an evolutionary trend toward TA systems with high expression levels of very
  \end{verbnobox}
  \end{document}